Question by Vince4815162342: Weed effects on arteries myth ?
My dad just told me that he was watching some biopsy cause he’s a cop and the doctor pointed at the mans arteries and said that they were worn down and weak because the man took medication, my dad then said that marijuana actually has the same effect, I felt like it was just a thing to make me not smoke marijuana but it sounds real don’t be a guy that just gets mad at society and stuff like that give me a medical answer that would be awesome

Best answer:

Answer by ricardo9505
“cough”
“bullsh%t”
“cough”

Answer by Metalplanttag
“We’ve done a number of clinical papers reporting on marijuana users,” Cadet said. “In the past, we have reported cognitive abnormalities, and a paper in 2005 reported vascular [blood vessel] abnormalities.” – http://health.usnews.com/health-news/family-health/articles/2008/05/13/marijuana-use-may-raise-risk-of-heart-attack-stroke

Cannabis use does have an effect on the brain and lungs:
Long-term heavy cannabis use in healthy individuals is associated with smaller cerebellar white-matter volume similar to that observed in schizophrenia. Reduced volumes were even more pronounced in patients with schizophrenia who use cannabis. Cannabis use may alter the course of brain maturational processes associated with schizophrenia. http://journals.cambridge.org/action/displayAbstract?fromPage=online&aid=8389504

Our findings indicate long-term cannabis use is hazardous to the white matter of the developing brain. Delaying the age at which regular use begins may minimize the severity of microstructural impairment. – http://brain.oxfordjournals.org/content/135/7/2245.short

Teens who routinely smoke marijuana risk a long-term drop in their IQ – http://news.yahoo.com/teen-pot-linked-later-declines-iq-192328332.html?_esi=0&bcmt=1346127366859-cc6205f3-31b2-4ebb-ac93-708fbfc37af9&bcmt_s=u#ugccmt-container-b

Informants also reported noticing more cognitive problems for persistent cannabis users. Impairment was concentrated among adolescent-onset cannabis users, with more persistent use associated with greater decline. Further, cessation of cannabis use did not fully restore neuropsychological functioning among adolescent-onset cannabis users. – http://www.pnas.org/content/early/2012/08/22/1206820109.abstract?sid=ca43e649-7ac1-4b18-94ac-ac62e23b6cea
These results provide new evidence of exposure-
related structural abnormalities in the hippocampus
and amygdala in long-term heavy cannabis users and
corroborate similar findings in the animal literature. These
findings indicate that heavy daily cannabis use across protracted
periods exerts harmful effects on brain tissue and
mental health. – http://www.truecompassion.org/PDFS/Health%20and%20Mental%20Health/Brain%20Regional%20Abnormalities%20Hippo%20Aust%20ArchGenPsych.pdf

Despite relatively brief exposure, adolescent
cannabis users relative to their age-matched counterparts
demonstrated similar memory deficits to those reported in
adult long-term heavy users. The results indicate that
cannabis adversely affects the developing brain and
reinforce concerns regarding the impact of early exposure. – http://www.acceptandchange.com/wp-content/uploads/2011/08/SolowijAdolescRAVLTPsychopharmacol.pdf

Marijuana abuse during pregnancy and adolescence represents a major health problem owing to its potential consequences on neural development. Prenatally cannabis-exposed children display cognitive deficits, suggesting that maternal consumption has interfered with the proper maturation of the brain. – http://www.futuremedicine.com/doi/full/10.2217/fnl.11.27
The smoke from marijuana, the second most
commonly smoked substance after tobacco, contains,
in addition to THC, a large number of toxic
gases and particulates (including high concentrations
of procarcinogenic polycyclic aromatic hydrocarbons)
that are capable of causing lung injury
and potentially respiratory malignancy. Whereas
THC produces short-term bronchodilation by relaxing
airway smooth muscle, heavy habitual use
of marijuana is associated with a number of adverse
pulmonary consequences (table 1). These include
1) symptoms of acute and chronic bronchitis,
2) conflicting findings concerning the presence
or absence of mild, progressive airflow obstruction,
3) endoscopic and microscopic evidence of
airway wall edema, vascular congestion and increased
mucous secretion, 4) extensive histopathologic
and immunohistochemical evidence of damage
and dysregulated growth of the tracheobronchial
epithelium, and 5) accumulation of increased
numbers of alveolar macrophages that
demonstrate impaired antimicrobial and tumoricidal
function and impaired ability to generate immunostimulatory
cytokines and inducible nitric
oxide synthase and nitric oxide, an important effector
molecule in microbial killing. These features
raise concern that marijuana smoking may be
a risk factor 1) for opportunistic infection, especially
in already immunocompromised patients
due to AIDS, organ transplanatation or cancer
chemotherapy, and 2) for upper and lower respiratory tract cancer. http://archest.fsm.it/pne/pdf/63/2/pne63-2_tashkin.pdf

bullous lung disease occurs in marijuana smokers approximately 20 years earlier than tobacco smokers. – http://www.sciencedaily.com/releases/2008/01/080123104017.htm

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